Bronchopulmonary dysplasia (BPD) is a common complication in newborn who require ventilatory therapy. The cumulative effects of oxygen exposure and the trauma of positive pressure ventilation in the immature lung are thought to be the primary causes [1]. Patients with BPD are cyanotic and oxygen-dependent, and have respiratory distress [2]. The clinical diagnosis of BPD is based on the facts that after 28 days of life there is a continuing need for oxygen and chest radiographic abnormalities persist [1].  An orderly progression of BPD through the four stages as first described by Northway and Rosan is uncommon [2,3]. Currently the severe cystic form of BPD is quite unusual and seen mostly in infants born very prematurely. Complications of BPD include tracheomalacia, tracheal stenosis, long-standing atelectasis, and acquired lobar emphysema [1].
        The findings of BPD on chest films vary, depending on the degree of focal overaeration of the alveoli, the degree of associated interstitial inflammatory change, and fibrosis [4,5]. These lead to overaeration of the lungs, diffuse haziness of the lungs, irregular infiltrates, streaks, or bubbles.